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Concern 01
Excessive Hair Fall
Losing more than 100 strands of hair per day is considered excessive. You may notice clumps in the shower drain, hair on your pillow, or significantly more shedding when combing. This is one of the most common concerns we treat at Aglow Hair — and one of the most successfully managed.
What Causes Excessive Hair Fall?
Telogen Effluvium — sudden physical or emotional stress pushes large numbers of hairs into the resting phase simultaneously. Common triggers: illness, surgery, rapid weight loss, relationship stress, or work burnout.
DHT Sensitivity (Androgenetic) — hereditary follicle sensitivity to dihydrotestosterone (DHT) causes progressive miniaturisation of follicles and increased daily shedding.
Nutritional Deficiency — iron deficiency (common in Malaysian women), low ferritin, zinc deficiency, and biotin insufficiency can all cause significant daily shedding.
Thyroid Dysfunction — both hyperthyroidism and hypothyroidism disrupt the hair growth cycle, causing diffuse shedding.
Scalp Inflammation — seborrhoeic dermatitis, fungal overgrowth, and chronic scalp inflammation physically damage follicles and accelerate shedding.
What our AI scan identifies
Follicle density, active vs resting follicle ratio, inflammation markers, sebum overproduction, and early miniaturisation patterns — giving your doctor a precise root cause before recommending treatment.
Recommended Treatment Path
Step 1: AI Scalp Scan (RM99) — identify root cause
Step 2: Scalp-Clarity or Scalp-Revival (RM299–RM399)
Step 3: LUMERA™ Laser LLLT (RM499) if DHT-driven
Step 4: Root-Fix™ or Stress-Shield™ oral (RM300–RM450)
If DHT-driven: PRP Plasma Shot (RM1,500) + DHT-Block™
Expected Timeline
Noticeable reduction in daily shed: 4–8 weeks. Visible density recovery: 8–16 weeks. Full programme results: 3–6 months.
Book AI Scan for Hair Fall
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Concern 02
Hair Thinning & Volume Loss
Hair feels flat. Your parting looks wider. The scalp is becoming increasingly visible through the hair. This is not the same as daily hair fall — thinning refers to the gradual reduction in the diameter and density of individual hair shafts, often caused by follicle miniaturisation over months or years.
Root Causes of Hair Thinning
Follicle Miniaturisation — hair follicles progressively shrink due to DHT, producing finer, shorter, lighter hairs until eventually ceasing production.
Hormonal Changes — reduced oestrogen (post-menopause) or elevated androgens shift the hair growth cycle balance toward shorter anagen phases.
Chronic Low-Grade Inflammation — persistent scalp inflammation damages follicles over time, often without obvious visible scalp symptoms.
Ageing Hair Cycle — naturally the anagen (growth) phase shortens with age, producing progressively thinner hair shafts from the same follicle.
Treatment Path for Thinning
Step 1: AI Scalp Scan — measure miniaturisation degree + terminal:vellus ratio
Step 2: LUMERA™ Laser + NÜROOTZ™ Red Light combo
Step 3: ScalpBoost Exosome Therapy (RM599)
Step 4: Plinest™ PN-HPT® or PRP injectables (RM1,500)
Daily: DHT-Block™ + Root-Core™ Minoxidil serum maintenance
Book Scan for Hair Thinning
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Concern 03 — Male
Male Pattern Baldness & Receding Hairline
Androgenetic alopecia in men — the classic M-shaped recession, crown thinning, or combined frontal and vertex loss. Caused by genetic DHT sensitivity. Affects approximately 50% of Malaysian men by age 50. The earlier treatment begins, the more follicles are preserved.
Understanding Male Pattern Loss
Norwood Scale I–VII: The standard classification for male pattern baldness severity. Our AI scan provides Norwood grading to guide your treatment pathway.
The DHT Mechanism: Hair follicles genetically programmed to be DHT-sensitive shrink progressively under hormone exposure — the only way to stop this is to reduce DHT levels or protect follicles from its effects.
Why It Starts at the Hairline: The frontal and vertex follicles are more DHT-sensitive than the sides and back — which is why FUE transplants use "donor hair" from the DHT-resistant back of the scalp.
Surgery vs Non-Surgical: Non-surgical options can slow recession and improve density significantly at Norwood I–III. Norwood IV+ may benefit from transplant consultation alongside medical management.
Male Pattern Treatment Path
Norwood I–II: LUMERA™ Laser + DHT-Block™/Hardcore™ oral + Root-Core™ Minoxidil serum
Norwood II–III: Above + PRP Plasma Shot at hairline (RM1,500) + ScalpBoost Exosome (RM599)
Norwood III+: Medical management + Hair Transplant (FUE) consultation
Key Message
Male pattern baldness is best treated early. Every month of untreated DHT exposure means more follicle miniaturisation that cannot be fully reversed.
Book Norwood Assessment
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Concern 04 — Female
Female Pattern Hair Loss (FPHL)
Female Pattern Hair Loss presents very differently to male baldness. Instead of a receding hairline, FPHL shows as a widening part, reduced hair volume from the crown, or a diffuse overall thinning. The hairline is typically preserved. It often starts after puberty or during hormonal transitions — menopause, post-pregnancy, or thyroid changes.
Causes of Female Pattern Hair Loss
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Androgen Sensitivity (Genetic): Like male pattern loss, some women have follicles genetically sensitive to DHT. This causes gradual miniaturisation, typically from the crown outward.
B
Reduced Oestrogen: Oestrogen protects follicles. Menopause, post-delivery hormonal drop, and certain contraceptive changes reduce this protection.
C
PCOS (Polycystic Ovary Syndrome): Elevated androgen levels in PCOS directly cause follicle miniaturisation in women — diagnosis requires blood testing alongside scalp scanning.
D
Thyroid Disorders: Both hypothyroidism and hyperthyroidism cause diffuse thinning in women — typically the entire scalp rather than crown-specific.
Female Pattern Treatment Path
Step 1: AI scan + Ludwig scale assessment + hormonal review discussion
Step 2: Scalp-Revival treatment + NÜROOTZ™ Red Light
Step 3: Plinest™ PN-HPT® injection (suitable for women) (RM1,500)
Step 4: Root-Fix™ or Stress-Shield™ oral based on root cause
Book Female Hair Loss Assessment
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Concern 05
Postpartum Hair Loss (After Childbirth)
One of the most distressing forms of hair loss — suddenly losing large amounts of hair 3–6 months after giving birth. This is extremely common and is caused by the dramatic drop in oestrogen levels post-delivery, which pushes a large portion of hairs into the shedding phase simultaneously. Most cases self-resolve — but treatment significantly accelerates recovery.
What You Need to Know
Normal — But Not Permanent: Postpartum hair loss is medically termed "telogen effluvium" and affects up to 50% of new mothers. It is temporary, but treatment cuts recovery time from 6–12 months to 2–4 months.
Timeline: Shedding peaks around 3–4 months post-delivery. Without treatment, most hair recovers by 6–12 months. With treatment: 2–4 months.
When to Be Concerned: If heavy shedding continues beyond 12 months, or if thinning is concentrated at the crown (not diffuse), an underlying condition like FPHL or thyroid issue should be ruled out via AI scan.
Safe for Breastfeeding Mothers: Our scalp therapy and red light treatments are fully safe for breastfeeding mothers. Some oral medications may require a pause in breastfeeding — this will be reviewed in your consultation.
Postpartum Treatment Path
Step 1: AI scan — confirm telogen effluvium vs underlying FPHL
Step 2: Scalp-Revival 2hr treatment (RM399) — scalp restoration + nutrient infusion
Step 3: Root-Fix™ oral nutritional support (RM300) — postpartum-specific formula
Step 4: NÜROOTZ™ Red Light (RM199) — follicle reactivation, safe for new mothers
Homecare: EquiScalp + Rootguard shampoo + Activ8 tonic daily
Book Postpartum Assessment
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Concern 06
Dandruff & Itchy Scalp
Persistent flaking, scalp itch, redness, or scalp sensitivity. Often caused by Malassezia yeast overgrowth, seborrhoeic dermatitis, or contact sensitivity. Malaysia's high humidity makes scalp conditions significantly worse than in temperate climates — fungal and bacterial overgrowth thrive in heat and moisture.
Types & Causes
1
Malassezia Yeast (Fungal Dandruff): The most common type. Yellow-white, oily flakes. Responds well to anti-fungal scalp treatment. Malaysia's humidity is the primary trigger.
2
Dry Scalp Dandruff: Small, white, dry flakes. Often caused by harsh shampoos stripping scalp lipids, over-washing, or low indoor humidity (air-conditioning).
3
Seborrhoeic Dermatitis: A chronic inflammatory condition causing redness, itching, and thick yellow scaling. Often extends to eyebrows and nasal folds. Requires medical management.
4
Contact Sensitivity: Reaction to hair dye, shampoo ingredients, or styling products. Often misdiagnosed as dandruff. AI scan reveals inflammation pattern to differentiate.
Treatment Path
Step 1: AI scan — identify fungal vs inflammatory vs dry scalp type
Step 2: Scalp-Clarity 1hr (RM299) — medicated deep cleanse + detox mask
Step 3: EquiScalp Balancing Shampoo daily (RM199)
If persistent: targeted anti-inflammatory ampoule sessions
Book Scalp Assessment
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Concern 07
Oily Scalp & Follicle Buildup
Excess sebum production makes hair greasy within hours of washing, causes scalp odour, and physically clogs hair follicles — blocking growth. A common concern in Malaysia given our tropical climate, and often a precursor to dandruff or accelerated hair loss if left untreated.
Why Oily Scalp Causes Hair Loss
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Follicle Blockage: Excess sebum mixes with dead skin cells and product residue to form a plug at the follicle opening — restricting hair shaft emergence and growth.
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Bacterial Growth: Sebum-rich scalp environments encourage bacterial proliferation, causing low-grade chronic scalp inflammation that damages follicles over time.
3
Hormonal Androgens: Elevated androgens (in both men and women) stimulate sebaceous glands to overproduce sebum — often the same hormones that drive hair loss.
Treatment Path
Step 1: AI scan — measure sebum production rate and follicle clogging grade
Step 2: Scalp-Revival deep detox (RM399) — sebum regulation + high-frequency therapy
Step 3: EquiScalp Balancing Shampoo + Activ8 Tonic daily homecare
If hormonal: DHT-Block™ review in consultation
Book Oily Scalp Assessment
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Concern 08
Alopecia Areata (Bald Patches)
Sudden, coin-shaped or irregular bald patches appearing on the scalp. Alopecia areata is an autoimmune condition where the immune system mistakenly attacks hair follicles. It can appear at any age, often triggered by stress or illness. Follicles remain alive in most cases — making early intervention highly effective.
Important Facts About Alopecia Areata
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Follicles Are Still Alive: Unlike scarring alopecia, the follicles in alopecia areata patches are usually dormant, not destroyed — meaning recovery is often possible with treatment.
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Early Treatment Has Highest Success Rate: The longer patches remain untreated, the more the immune attack damages the follicle base. Treat within 3–6 months of appearance for best outcomes.
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AI Scan Identifies Patch Stage: Exclamation mark hairs (broken hairs at patch edges) confirm active inflammation. Peach fuzz regrowth (vellus hairs) at the patch indicates the follicle is recovering.
Alopecia Areata Treatment Path
Step 1: AI scan — confirm AA type and assess follicle viability
Step 2: LUMERA™ Laser LLLT at patch sites (RM499) — anti-inflammatory reactivation
Step 3: PRP Plasma Shot at patches (RM1,500) — growth factor stimulation
Step 4: Plinest™ PN-HPT® injection — cellular regeneration at follicle level
Book Alopecia Assessment